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Devic's syndrome

Devic’s syndrome, more commonly known as Neuromyelitis Optica Spectrum Disorder (NMOSD), is a rare autoimmune inflammatory disease of the central nervous system.

Prevalence

1 / 100 000

1–4 per 100,000

US Estimated

0.5–2 per 100,000

Europe Estimated

Age of Onset

Adulthood

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ICD-10

G36.0

Inheritance Pattern

Autosomal dominant

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Autosomal recessive

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Mitochondrial/Multigenic

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X-linked dominant

Rare

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X-linked recessive

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5 Facts you should know

FACT

1

Devic's Syndrome, also known as Neuromyelitis Optica (NMO), is a rare autoimmune disorder primarily affecting the optic nerves and spinal cord, causing inflammation and demyelination

FACT

2

It is characterized by recurrent attacks of optic neuritis, leading to visual impairment or blindness, and transverse myelitis, resulting in weakness, sensory loss, and sometimes paralysis of the limbs

FACT

3

NMO is associated with autoantibodies against aquaporin-4 (AQP4), found abundantly in the optic nerves and spinal cord, contributing to immune-mediated damage

FACT

4

Differential diagnosis involves distinguishing NMO from multiple sclerosis (MS) due to similarities in symptoms, but NMO typically presents with more severe optic neuritis and longitudinally extensive transverse myelitis

FACT

5

Treatment includes immunosuppressive therapies such as corticosteroids, plasma exchange, and targeted biologics like rituximab or eculizumab to manage attacks and prevent relapses

Devic’s syndrome is also known as...

Devic’s syndrome is also known as:

  • Neuromyelitis optica (NMO)
  • Neuromyelitis optica spectrum disorder (NMOSD)

What’s your Rare IQ?

Devic's Syndrome, also known as Neuromyelitis Optica (NMO), is a rare autoimmune disorder affecting the central nervous system. What is a characteristic feature of Devic's Syndrome?

 

Common signs & symptoms

Optic neuritis with vision loss or eye pain

Transverse myelitis causing limb weakness or paralysis

Sensory disturbances

Bladder and bowel dysfunction

Severe nausea, vomiting, or hiccups (area postrema syndrome)

Recurrent attacks with incomplete recovery

Current treatments

Acute attack management:

  • High-dose intravenous corticosteroids
  • Plasma exchange (PLEX) for steroid-refractory attacks

Long-term disease-modifying therapies:

  • Immunosuppressive agents (e.g., azathioprine, mycophenolate mofetil)
  • Targeted biologic therapies approved for NMOSD, including complement and B-cell pathway inhibitors

References:

Wingerchuk DM, Banwell B, Bennett JL, et al. International Panel for NMO Diagnosis. International consensus diagnostic criteria for neuromyelitis optica spectrum disorders. <i>Neurology.</i> 2015;85(2):177–189. doi:10.1212/WNL.0000000000001729 Pittock SJ, Lucchinetti CF, Lennon VA, et al. Efficacy and safety of eculizumab in neuromyelitis optica spectrum disorder. <i>N Engl J Med.</i> 2019;381(7):614–625. doi:10.1056/NEJMoa1900866 Jarius S, Ruprecht K, Kleiter I, et al. Neuromyelitis optica spectrum disorders: update on diagnosis, pathophysiology, and treatment. <i>Curr Opin Neurol.</i> 2016;29(3):276–283. doi:10.1097/WCO.0000000000000324 Huda S, Whittam DH, Bhojak M, et al. Neuromyelitis optica spectrum disorders: review of clinical features, diagnostic criteria and treatment options. <i>Clin Exp Immunol.</i> 2019;195(2):149–164. doi:10.1111/cei.13295 Pittock SJ, Weinshenker BG, Lucchinetti CF, et al. Neuromyelitis optica and non organ‐specific autoimmunity. <i>Arch Neurol.</i> 2004;61(2):166–171. doi:10.1001/archneur.61.2.166